CBL: Transient Monocular Vision Loss in a 71-Year-Old
A retired teacher with brief, recurrent greying of the left eye.
Learning Objectives
- Recognize amaurosis fugax as transient monocular vision loss (TMVL).
- Understand that AFx is a medical emergency requiring urgent carotid/vascular evaluation.
- Differentiate ocular, optic nerve, and central causes; consider GCA workup in the appropriate demographic.
Vignette
A 71-year-old retired kindergarten teacher reports 1–2 weeks of transient greying haze over the left eye, lasting minutes and resolving completely. No headache during episodes; denies jaw claudication, scalp tenderness, myalgias, or joint pain. Medications: lisinopril, aspirin, simvastatin, omeprazole, tolterodine. PMH: hypertension, hyperlipidemia, GERD, urinary incontinence, hysterectomy, tonsillectomy. Social: never smoker, ~2 glasses wine/week, lives alone. Family history: lupus (daughter), Alzheimer disease (mother), non-Hodgkin lymphoma (father). ROS otherwise unremarkable; vision currently normal.
Exam: Pulse 82, BP 131/83, afebrile. Cardiovascular exam normal; carotid auscultation without bruits. Cranial nerves intact; visual acuity corrected to baseline; pupils and fields normal; extraocular movements intact. Strength and tone normal; sensation intact; reflexes normal except absent Achilles bilaterally; plantar responses downgoing; coordination and gait normal; negative Romberg.
Localization and Neuroanatomy
The episodic, negative visual phenomenon ("greying out") affecting a single eye for minutes localizes anterior to the chiasm—retina, optic nerve, or ocular perfusion. Lack of positive visual phenomena argues against migraine aura. Transient monocular loss suggests retinal/optic nerve ischemia (retinal TIA) most commonly from ipsilateral internal carotid atherosclerotic disease with distal embolization.
Diagnosis
Treat as a medical emergency similar to TIA. Urgent vascular imaging of the head/neck (carotid duplex ultrasound, CTA, or MRA) is indicated to evaluate for carotid stenosis/plaques. Prompt ophthalmic evaluation helps exclude retinal pathology; check intraocular pressure. In patients >50 with concerning features for giant cell arteritis (GCA)—new headache, jaw claudication, scalp tenderness, polymyalgia—obtain ESR/CRP and CBC and consider temporal artery ultrasound/biopsy.
Question 1
Amaurosis fugax most directly reflects transient ischemia of which structure?
- A. Brainstem
- B. Retina/optic nerve of the eye
- C. Spinal cord
- D. Myocardium
Question 2
Which is the most appropriate urgent diagnostic step?
- A. MRI brain only
- B. Carotid vascular imaging (duplex, CTA, or MRA)
- C. Neck CT without contrast
- D. Electrophysiologic testing
Question 3
When carotid imaging shows ipsilateral symptomatic stenosis, what is the preferred management?
- A. Blood pressure support
- B. Maximize medical therapy only regardless of stenosis
- C. Carotid endarterectomy (CEA) or stenting for high-grade stenosis plus best medical therapy
- D. Ophthalmologic surgery
Teaching Points
- Amaurosis fugax = transient monocular negative visual loss (retinal TIA) and is a stroke warning.
- Urgent carotid imaging (duplex/CTA/MRA) is recommended; evaluate the eye as well (retina, IOP).
- Consider GCA in patients ≥50 with suggestive symptoms; obtain ESR/CRP and arrange imaging/biopsy if indicated.
- For symptomatic carotid stenosis 70–99%, CEA (or CAS in appropriate patients) plus best medical therapy reduces stroke risk; optimize antiplatelet, statin, BP, and lifestyle.
References
- AHA/ASA. Prevention of Stroke in Patients With Stroke and TIA. Stroke. 2021.
- ESVS. 2023 Guidelines on Atherosclerotic Carotid & Vertebral Disease.
- StatPearls. Amaurosis Fugax (updated 2025).
- ACR/EULAR 2022 GCA Classification Criteria.
- AAO EyeNet. Retinal TIAs: A Medical Emergency.